In vivo base editing of PCSK9 with VERVE-102 for hypercholesterolemia (Registro nro. 22825)

000 -Cabecera
Campo de control interno 02909nam a2200301 4500
001 - Número de control
control field ESSALUD
005 - Fecha y hora de la última transacción
Campo de control 20260817160341.0
007 - Tipo material - Descripcion fisica - info general
Tipo material - Descripcion fisica - info general ta
008 - Elementos de Longitud Fija - Información General
Elementos de Longitud Fija - Información t pe ||||| |||| 00| 0 spa d
040 ## - Origen de la Catalogación
Origen de la Catalogación BMG
041 ## - Idioma
Idioma eng
100 ## - Autor
Autor Vafai, Scott B.
Rol del Autor Autor
9 (RLIN) 54105
245 ## - Titulo
Titulo In vivo base editing of PCSK9 with VERVE-102 for hypercholesterolemia
300 ## - Páginas
Paginación páginas: 648-659
520 ## - Resumen
Resumen Background: Persons carrying loss-of-function variants of proprotein convertase subtilisin–kexin type 9 (PCSK9) have reduced levels of low-density lipoprotein (LDL) cholesterol and fewer atherosclerotic cardiovascular disease events than persons without such variants. VERVE-102 is an investigational base-editing therapy designed to durably inactivate PCSK9 in the liver. Methods: In this phase 1, open-label, single-ascending-dose study, we administered one intravenous infusion of VERVE-102 at one of six doses (ranging from 0.3 to 1.0 mg of total RNA per kilogram of body weight [mg per kilogram]) to adults with heterozygous familial hypercholesterolemia or premature coronary artery disease. VERVE-102 consists of a messenger RNA encoding an adenine base-editor protein and a guide RNA targeting PCSK9, which are encapsulated in a lipid nanoparticle incorporating N-acetylgalactosamine. The objectives were to assess safety and changes in blood PCSK9 protein and LDL cholesterol levels. Results: A total of 35 participants across the six dose cohorts received VERVE-102 and had at least 28 days of follow-up. No dose-limiting toxic effects occurred. Mild-to-moderate infusion-related reactions and transient elevations in alanine aminotransferase levels were observed. Aspiration pneumonitis occurred in a participant with gastroesophageal reflux disease. Dose-dependent mean reductions in the PCSK9 level ranged from 51% at the 0.3-mg-per-kilogram dose to 88% at the 1.0-mg-per-kilogram dose. Corresponding reductions in the LDL cholesterol level ranged from 9% at the 0.3-mg-per-kilogram dose to 62% at the 1.0-mg-per-kilogram dose, with an absolute reduction of 78 mg per deciliter at the highest dose. Reductions appeared to be durable throughout follow-up, which was at least 1 year in 15 participants. Conclusions: One dose of VERVE-102 led to dose-dependent, substantial, and sustained reductions in PCSK9 and LDL cholesterol levels.
650 ## - Temas - Descriptores
Temas - Descriptores CARDIOLOGÍA GENERAL
9 (RLIN) 53638
650 ## - Temas - Descriptores
Temas - Descriptores EDICIÓN GENÉTICA
9 (RLIN) 54106
650 ## - Temas - Descriptores
Temas - Descriptores GENÉTICA GENERAL
9 (RLIN) 54107
650 ## - Temas - Descriptores
Temas - Descriptores LÍPIDOS
9 (RLIN) 51326
700 ## - Autor Personal
Autor Personal Täubel, Jörg
9 (RLIN) 54108
700 ## - Autor Personal
Autor Personal Ashdown, Thomas
9 (RLIN) 54109
700 ## - Autor Personal
Autor Personal Patel, Riyaz S.
9 (RLIN) 54110
700 ## - Autor Personal
Autor Personal Diamondali, Sadaf
9 (RLIN) 54111
700 ## - Autor Personal
Autor Personal Cegla, Jaimini
9 (RLIN) 54112
700 ## - Autor Personal
Autor Personal Soran, Handrean
9 (RLIN) 54113
773 0# - Revista (Relacion con el numero)
Host Biblionumber 22717
Host Itemnumber 22647
Ciudad, Editorial Massachusetts NEJM Group
Codigo barras item/ejemplar NEJM010
Titulo de la Revista The New England Journal of Medicine
Número de control de registro ESSALUD
ISSN 0028-4793
942 ## - Elementos de Koha
Tipo de Documento Artículos
Fecha procesamiento 2026-08-17
Catalogador SQB

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